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Considerations for the Diagnosis of Chemical Sensitivity

Posted on January 19, 2022 at 4:25 AM


 

William J. Rea Alfred R. Johnson, Gerald H. Ross, Joel R. Butler, Ervin J. Fenyves, Bertie Griffiths, and John Laseter

 



Introduction


The study of the effects of the environment upon the individual is now feasible due to new technology developed in the construction of environmental units.1,2,3. Our observations reveal that individual or multiple organs may be involved. The brain is the target organ in only a subset of chemically sensitive patients, and its involvement should not be confused with psychosomatic disease.

 

Over the last 16 years physicians and scientists at the Environmental Health Center in Dallas have had an opportunity to observe over 20,000 patients who had chemical sensitivity problems. These patients were studied under various degrees of environmental control. This experience is unique in the world and has resulted in numerous peer-reviewed scientific articles, chapters in books, and books on this subject.

 

Studies have resulted in over 32,000 challenge tests by inhalation, oral, or injection methods, of which 16,000 are double-blind. Blood chemical levels and fat biopsies for organic hydrocarbons number over 2,000, while the measurement of immune parameters are over 5,000 tests. Objective brain function tests have been accomplished in over 5,000 patients. Other objective tests, like computerized balance studies, depollutant enzyme levels, and autonomic nervous system changes as measured by the Iriscorder, number near 1,000.

 

We wish to share our findings with the participants of the National Academy of Sciences Committee for the study of chemical sensitivity.



 

Definition and Principles


Chemical sensitivity is defined as an adverse reaction to ambient doses of toxic chemicals in our air, food, and water at levels which are generally accepted as subtoxic. Manifestation of adverse reactions depend on: (1) the tissue or organ involved; (2) the chemical and pharmacologic nature of the toxin; (3) the individual susceptibility of the exposed person (genetic make-up, nutritional state, and total load at the time of exposure); (4) the length of time of the exposure; (5) mount and variety of other body stressors (total load) and synergism at the time of reaction. (6) the derangement of metabolism that may occur from the initial insults.

 

To demonstrate cause-and-effect proof of environmental influence on an individual's health, one must understand several important principles and facts. These principles involve those of total body load (burden), adaptation (masking, acute toxicological tolerance), bipolarity, biochemical individuality. Each principle will be discussed separately.........


Indexed for NIH by Dragonfly Kingdom Library

Full document at https://www.ncbi.nlm.nih.gov/books/NBK234807/


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